| [1]Di Benedetto A, Gigante I, Colucci S, et al.Periodontal disease: linking the primary inflammation to bone loss[J].Clin Dev Immunol,2013,2013:503754-.[2]Fujihashi K, Yamamoto M, Hiroi T, et al.Selected Th1 and Th2 cytokine mRNA expression by CD4(+) T cells isolated from inflamed human gingival t3s[J].Clin Exp Immunol,1996,103(3):422-428.[3]Noack M, Miossec P.Th17 and regulatory T cell balance in autoimmune and inflammatory diseases[J].Autoimmun Rev,2014,13(6):668-677.[4]Kaur S, White S, Bartold PM.Periodontal disease and rheumatoid arthritis: a systematic review[J].J Dent Res,2013,92(5):399-408.[5]Cardoso CR, Garlet GP, Crippa GE, et al.Evidence of the presence of T helper type 17 cells in chronic lesions of human periodontal disease[J].Oral Microbiol Immunol,2009,24(1):1-6.[6]Gemmell E1, Yamazaki K, Seymour GJ.Destructive periodontitis lesions are determined by the nature of the lymphocytic response[J].Crit Rev Oral Biol Med,2002,13(1):17-34.[7]Dutzan N, Gamonal J, Silva A, et al.Over-expression of forkhead box P3 and its association with receptor activator of nuclear factor-kappa B ligand, interleukin (IL) -17, IL-10 and transforming growth factor-beta during the progression of chronic periodontitis[J].J Clin Periodontol,2009,36(5):396-403.[8]Cheng WC, Hughes FJ, Taams LS.The presence, function and regulation of IL-17 and Th17 cells in periodontitis[J].J Clin Periodontol,2014,41(6):541-549.[9]Zhao L, Zhou Y, Xu Y, et al.Effect of non-surgical periodontal therapy on the levels of Th17/Th1/Th2 cytokines and their transcription factors in Chinese chronic periodontitis patients[J].J Clin Periodontol,2011,38(6):509-516.[10]Moutsopoulos NM, Kling HM, Angelov N, et al.Porphyromonas gingivalis promotes Th17 inducing pathways in chronic periodontitis[J].J Autoimmun,2012,39(4):294-303.[11]Behfarnia P, Birang R, Pishva SS, et al.Expression levels of th-2 and th-17 characteristic genes in healthy t1 versus periodontitis[J].J Dent (Tehran),2013,10(1):23-31.[12]Fu QY, Zhang L, Duan L, et al.Correlation of chronic periodontitis in tropical area and IFN-γ, IL-10, IL-17 levels[J].Asian Pac J Trop Med,2013,6(6):489-492.[13]Sakaguchi S.Naturally arising CD4+ regulatory t cells for immunologic self-tolerance and negative control of immune responses[J].Annu Rev Immunol,2004,22:531-562.[14]Cardoso CR, Garlet GP, Moreira AP, et al.Characterization of CD4+CD25+ natural regulatory T cells in the inflammatory infiltrate of human chronic periodontitis[J].J Leukoc Biol,2008,84(1):311-318.[15]Nakajima T, Ueki-Maruyama K, Oda T, et al.Regulatory T-cells infiltrate periodontal disease t7s[J].J Dent Res,2005,84(7):639-643.[16]Garlet GP, Cardoso CR, Mariano FS, et al.Regulatory T cells attenuate experimental periodontitis progression in mice[J].J Clin Periodontol,2010,37(7):591-600.[17]Glowacki AJ, Yoshizawa S, Jhunjhunwala S, et al.Prevention of inflammation-mediated bone loss in murine and canine periodontal disease via recruitment of regulatory lymphocytes[J].Proc Natl Acad Sci U S A,2013,110(46):18525-18530.[18]Ohlrich EJ, Cullinan MP, Seymour GJ.The immunopathogenesis of periodontal disease[J].Aust Dent J,2009,54 (Suppl 1):S2-S10.[19]Bettelli E, Carrier Y, Gao W, et al.Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells[J].Nature,2006,441(7090):235-238.[20]Wang H, Peng W, Weng Y, et al.Imbalance of Th17/Treg cells in mice with chronic cigarette smoke exposure[J].Int Immunopharmacol,2012,14(4):504-512.[21]Ebersole JL, Kirakodu S, Novak MJ,et al .Cytokine gene expression profiles during initiation, progression and resolution of periodontitis[J].J Clin Periodontol,2014,41(9):853-861.[22]Ohyama H, Kato-Kogoe N, Kuhara A, et al.The involvement of IL-23 and the Th17 pathway in periodontitis[J].J Dent Res,2009,88(7):633-638.[23]Yang S, Zhu L, Xiao L, et al.Imbalance of interleukin-17+ T-cell and Foxp3+ regulatory T-cell dynamics in rat periapical lesions[J].J Endod,2014,40(1):56-62.[24]Ernst CW, Lee JE, Nakanishi T, et al.Diminished forkhead box P3/CD25 double-positive T regulatory cells are associated with the increased nuclear factor-kappaB ligand (RANKL+) T cells in bone resorption lesion of periodontal disease[J].Clin Exp Immunol,2007,148(2):271-280. |
