[1]Undale AH, Westendorf JJ, Yaszemski MJ, et al.Mesenchymal stem cells for bone repair and metabolic bone diseases[J].Mayo Clin Proc,2009,84(10):893-902.[2]Bradley EW, McGee-Lawrence ME, Westendorf JJ.Hdac-mediated control of endochondral and intramembranous ossification[J].Crit Rev Eukaryot Gene Expr,2011,21(2):101-113.[3]Teven CM, Liu X, Hu N, et al.Epigenetic regulation of mesenchymal stem cells: a focus on osteogenic and adipogenic differentiation[J].Stem Cells Int,2011,2011:201371-.[4]Haberland M, Mokalled MH, Montgomery RL, et al.Epigenetic control of skull morphogenesis by histone deacetylase 8[J].Genes Dev,2009,23(14):1625-1630.[5]Khan O, La Thangue NB.HDAC inhibitors in cancer biology: emerging mechanisms and clinical applications[J].Immunol Cell Biol,2012,90(1):85-94.[6]Schroeder TM, Westendorf JJ.Histone deacetylase inhibitors promote osteoblast maturation[J].J Bone Miner Res,2005,20(12):2254-2263.[7]Svingen T, Wilhelm D, Combes AN, et al.Ex vivo magnetofection: a novel strategy for the study of gene function in mouse organogenesis[J].Dev Dyn,2009,238(4):956-964.[8]Hassig CA, Schreiber SL.Nuclear histone acetylases and deacetylases and transcriptional regulation: HATs off to HDACs[J].Curr Opin Chem Biol,1997,1(3):300-308.[9]Glozak MA, Sengupta N, Zhang X, et al.Acetylation and deacetylation of non-histone proteins[J].Gene,2005,363:15-23.[10]Yan W, Liu S, Xu E, et al.Histone deacetylase inhibitors suppress mutant p53 transcription via histone deacetylase 8[J].Oncogene,2013,32(5):599-609.[11]Vanhaecke T, Papeleu P, Elaut G, et al.Trichostatin A-like hydroxamate histone deacetylase inhibitors as therapeutic agents: toxicological point of view[J].Curr Med Chem,2004,11(12):1629-1643.[12]何静, 刘红利, 陈燕.Effects of trichostatin A on HDAC8 expression, proliferation and cell cycle of Molt-4 cells[J].华中科技大学学报(医学英德文版),2006,26(5):531-533.[13]傅瑜,张平,张永栋,等.HDAC8过表达慢病毒载体构建、转染及表达的研究[J].口腔生物医学,2012,3(4):181-184. |