| [1]Mysak J1, Podzimek S1, Sommerova P1, et al.Porphyromonas gingivalis: major periodontopathic pathogen overview[J].J Immunol Res,2014,2014:476068-.[2]Runyen-Janecky LJ.Role and regulation of heme iron acquisition in gram-negative pathogens[J].Front Cell Infect Microbiol,2013,3:55-.[3]Gao JL1, Lu Y, Browne G, et al.The role of heme binding by DNA-protective protein from starved cells (Dps) in the Tolerance of Porphyromonas gingivalis to heme toxicity[J].J Biol Chem,2012,287(50):42243-42258.[4]Henry LG1, McKenzie RM, Robles A, et al.Oxidative stress resistance in Porphyromonas gingivalis[J].Future Microbiol,2012,7(4):497-512.[5]Johnson NA1, McKenzie RM, Fletcher HM.The bcp gene in the bcp-recA-vimA-vimE-vimF operon is important in oxidative stress resistance in Porphyromonas gingivalis W83[J].Mol Oral Microbiol,2011,26(1):62-77.[6]Dashper SG1, Ang CS, Veith PD,et al.Response of Porphyromonas gingivalis to heme limitation in continuous culture[J].J Bacteriol,2009,191(3):1044-1055.[7]Weinberg ED.Iron availability and infection[J].Biochim Biophys Acta,2009,1790(7):600-605.[8]Xie H1, Zheng C.OxyR activation in Porphyromonas gingivalis in response to a hemin-limited environment[J].Infect Immun,2012,80(10):3471-3480.[9]Hiratsuka K1, Kiyama-Kishikawa M, Abiko Y.Hemin-binding protein 35 (HBP35) plays an important role in bacteria-mammalian cells interactions in Porphyromonas gingivalis[J].Microb Pathog,2010,48(3-4):116-123.[10]P?ll?nen MT1, Paino A, Ihalin R.Environmental stimuli shape biofilm formation and the virulence of periodontal pathogens[J].Int J Mol Sci,2013,14(8):17221-17237.[11]Xie H1, Cai S, Lamont RJ.Environmental regulation of fimbrial gene expression in Porphyromonas gingivalis[J].Infect Immun,1997,65(6):2265-2271.[12]Kesavalu L1, Holt SC, Ebersole JL.In vitro environmental regulation of Porphyromonas gingivalis growth and virulence[J].Oral Microbiol Immunol,2003,18(4):226-233.[13]McKee AS, McDermid AS, Baskerville A, et al.Effect of hemin on the physiology and virulence of Bacteroides gingivalis W50[J].Infect Immun,1986,52(2):349-355.[14]Masuda T1, Murakami Y, Noguchi T, et al.Effects of various growth conditions in a chemostat on expression of virulence factors in Porphyromonas gingivalis[J].Appl Environ Microbiol,2006,72(5):3458-3467.[15]Al-Qutub MN1, Braham PH, Karimi-Naser LM, et a1.Hemin-dependent modulation of the lipid A structure of Porphyromonas gingivalis lipopolysaccharide[J].Infect lmmun,2006,74(8):4474-4485.[16]Curtis MA1, Percival RS, Devine D, et al.Temperature-dependent modulation of Porphyromonas gingivalis lipid A structure and interaction with the innate host defenses[J].Infect Immun,2011,79(3):1187-1193.[17]冯琴，张凤秋.牙龈蛋白酶及其致病作用[J].国际口腔医学杂志,2012,39(6):794-796.[18]张瑞，孙卫斌.牙龈素在牙龈卟啉单胞菌生长及牙周致病过程中的作用[J].口腔生物医学,2014,5(2):100-102. |
