[1]Chang ZL.Recent development of the mononuclear phagocyte system: in memory of Metchnikoff and Ehrlich on the 100th Anniversary of the 1908 Nobel Prize in Physiology or Medicine[J].Biol Cell,2009,101(12):709-721.[2]Gordon S.Elie Metchnikoff: father of natural immunity[J].Eur J Immunol,2008,38(12):3257-3264.[3]van Furth R, Raeburn JA, van Zwet TL.Characteristics of human mononuclear phagocytes[J].Blood,1979,54(2):485-500.[4]Grage-Griebenow E, Lorenzen D, Fetting R, et al.Phenotypical and functional characterization of Fc gamma receptor I (CD64)-negative monocytes, a minor human monocyte subpopulation with high accessory and antiviral activity[J].Eur J Immunol,1993,23(12):3126-3135.[5]Arinobu Y, Akashi K.Myeloid lineage commitment from the hematopoietic stem cell[J].Arerugi,2011,60(7):817-822.[6]Sunderk?tter C, Nikolic T, Dillon MJ, et al.Subpopulations of mouse blood monocytes differ in maturation stage and inflammatory response[J].J Immunol,2004,172(7):4410-4417.[7]Herwald H, Egesten A.Macrophages: past, present and future[J].J Innate Immun,2013,5(6):657-658.[8]Landsman L, Varol C, Jung S.Distinct differentiation potential of blood monocyte subsets in the lung[J].J Immunol,2007,178(4):2000-2007.[9]Krausgruber T, Blazek K, Smallie T, et al.IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses[J].Nat Immunol,2011,12(3):231-238.[10]Whyte CS, Bishop ET, Rückerl D, et al.Suppressor of cytokine signaling (SOCS)1 is a key determinant of differential macrophage activation and function[J].J Leukoc Biol,2011,90(5):845-854.[11]Nahrendorf M, Swirski FK, Aikawa E, et al.The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions[J].J Exp Med,2007,204(12):3037-3047.[12]Arnold L, Henry A, Poron F, et al.Inflammatory monocytes recruited after skeletal muscle injury switch into antiinflammatory macrophages to support myogenesis[J].J Exp Med,2007,204(5):1057-1069.[13]Schwartz M."T7-repairing" blood-derived macrophages are essential for healing of the injured spinal cord: from skin-activated macrophages to infiltrating blood-derived cells?[J].Brain Behav Immun,2010,24(7):1054-1057.[14]Landsman L, Bar-On L, Zernecke A, et al.CX3CR1 is required for monocyte homeostasis and atherogenesis by promoting cell survival[J].Blood,2009,113(4):963-972.[15]De Palma M, Venneri MA, Galli R, et al.Tie2 identifies a hematopoietic lineage of proangiogenic monocytes required for tumor vessel formation and a mesenchymal population of pericyte progenitors[J].Cancer Cell,2005,8(3):211-226.[16]Steinman RM, Witmer MD.Lymphoid dendritic cells are potent stimulators of the primary mixed leukocyte reaction in mice[J].Proc Natl Acad Sci U S A,1978,75(10):5132-5136.[17]De Smedt T, Pajak B, Muraille E, et al.Regulation of dendritic cell numbers and maturation by lipopolysaccharide in vivo[J].J Exp Med,1996,184(4):1413-1424.[18]Serbina NV, Salazar-Mather TP, Biron CA, et al.TNF/iNOS-producing dendritic cells mediate innate immune defense against bacterial infection[J].Immunity,2003,19(1):59-70.[19]Nakano H, Lin KL, Yanagita M, et al.Blood-derived inflammatory dendritic cells in lymph nodes stimulate acute T helper type 1 immune responses[J].Nat Immunol,2009,10(4):394-402.[20]Nagasawa T, Kobayashi H, Aramaki M, et al.Expression of CD14, CD16 and CD45RA on monocytes from periodontitis patients[J].J Periodontal Res,2004,39(1):72-78.[21]Andreesen R, Brugger W, Scheibenbogen C, et al.Surface phenotype analysis of human monocyte to macrophage maturation[J].J Leukoc Biol,1990,47(6):490-497.[22]Mackensen A, Galanos C, Wehr U, et al.Endotoxin tolerance: regulation of cytokine production and cellular changes in response to endotoxin application in cancer patients[J].Eur Cytokine Netw,1992,3(6):571-579.[23]Belge KU, Dayyani F, Horelt A, et al.The proinflammatory CD14+CD16+DR++ monocytes are a major source of TNF[J].J Immunol,2002,168(7):3536-3542.[24]Ebersole JL, Machen RL, Steffen MJ, et al.Systemic acute-phase reactants, C-reactive protein and haptoglobin, in adult periodontitis[J].Clin Exp Immunol,1997,107(2):347-352.[25]Beck JD, Offenbacher S.The association between periodontal diseases and cardiovascular diseases: a state-of-the-science review[J].Ann Periodontol,2001,6(1):9-15.[26]Kanaya S, Nemoto E, Ogawa T, et al.Porphyromonas gingivalis lipopolysaccharides induce maturation of dendritic cells with CD14+CD16+ phenotype[J].Eur J Immunol,2004,34(5):1451-1460.[27]Torsteinsdóttir I, Arvidson NG, H?llgren R, et al.Monocyte activation in rheumatoid arthritis (RA): increased integrin, Fc gamma and complement receptor expression and the effect of glucocorticoids[J].Clin Exp Immunol,1999,115(3):554-560.[28]Nicu EA, van der Velden U, Everts V, et al.Expression of FcgammaRs and mCD14 on polymorphonuclear neutrophils and monocytes may determine periodontal infection[J].Clin Exp Immunol,2008,154(2):177-186. |